James W. Kronstad

This study compares gene expression profiles of two spore types in Colletotrichum graminicola during development and infection, revealing distinct transcriptomes and functional differences.

This paper identifies a fungal effector, UmPce3, that targets the chloroplast DEAD-box RNA helicase RH3 to manipulate chloroplast function, enhancing fungal pathogenesis while balancing plant stress responses.

The paper identifies butyrolactol A as a compound that enhances caspofungin efficacy against resistant fungi by inhibiting phospholipid flippase, thereby improving antifungal treatment.

This study shows that polyphosphate mobilization is critical for Cryptococcus neoformans virulence in a mouse model, affecting cell surface structure, immune response, and mitochondrial function.

This study shows that glutathione metabolism in Cryptococcus neoformans affects virulence by modulating redox processes and melanin production in a murine infection model.

This paper identifies brilacidin as a novel antifungal agent effective against Cryptococcus neoformans, showing it reduces fungal survival in macrophages and potentiates existing antifungals.

This paper investigates how mitochondrial electron transport chain function and iron regulatory factors influence melanin formation in Cryptococcus neoformans, revealing a link between mitochondrial dysfunction, redox homeostasis, and virulence.

This study identifies key pathways regulating growth and virulence in Verticillium dahliae, highlighting the unfolded protein response, pheromone MAP kinase pathway, and oxylipin precursor formation as critical for pathogenesis.

This study identifies media-dependent secretome responses in Verticillium longisporum, revealing specific exoproteome patterns in plant-related environments and identifying Nlp2 and Nlp3 as key effectors for plant infection.

The paper uses conditional gene expression to study the unfolded protein response (UPR) in Ustilago maydis during plant infection, showing its role in suppressing host defense and preventing hyperactivation.