Steven Williams

The paper examines longitudinal changes in fronto-striatal glutamate and its relationship with inhibitory control functioning in ASD and OCD across a one-year period.

The paper identifies atypical brain asymmetry in autism as a potential neurophenotype for stratification, showing individualized patterns linked to symptom severity and language delay.

This study examines functional connectivity changes during and after cold pain in healthy volunteers, finding persistent alterations related to reported pain intensity.

This study investigates the role of TLR2 in neuroinflammatory responses in a rodent model of obstructive sleep apnea, linking it to behavioral and metabolic changes.

The paper develops a phenotypic stratification model to predict autism subtypes based on SC-RRB balance and applies it to fMRI data, identifying subtype-specific neural circuitry and gene expression patterns.

This study compares intranasal and intravenous oxytocin administration effects on regional cerebral blood flow, showing systemic OT increases drive amygdala perfusion changes and validating intranasal targeting of specific brain regions.

This study shows that effective cognitive therapy for PTSD restores balance in brain networks involved in self-referential processing and threat detection.

This study identifies functional connectivity differences in the posteromedial cortex across Alzheimer’s disease stages using constrained ICA and ADNI data.

This study uses resting-state functional imaging meta-analysis to find shared and distinct brain activation patterns between borderline personality disorder and post-traumatic stress disorder.

This study shows that delayed peripheral infusion of neurotrophin-3 improves sensorimotor recovery after ischemic stroke in rats by inducing spinal neuroplasticity.

The paper investigates how cerebral blood flow changes in response to drugs targeting different neurotransmitter systems and finds a reliable spatial link between blood flow changes and receptor densities.

This study identifies volumetric grey matter changes in very preterm individuals from adolescence to adulthood, showing accelerated brain maturation and regional vulnerability.