Eli Boritz

The paper models how delayed immune responses and prolonged infections increase the transmission risk of SARS-CoV-2 variants with fitness-enhancing mutations.

This study uses DAb-seq to analyze HIV-infected CD4 T cells on ART, revealing subset-specific differences in provirus maintenance and surface protein expression.

The paper identifies distinct follicular helper CD4 T cell programming in HIV-positive individuals who produce broadly neutralizing antibodies, linking it to improved B cell follicle architecture and reduced suppressor cells.

This study shows that advanced HIV infection leads to high intra-host SARS-CoV-2 spike diversity and adaptive evolution, potentially contributing to new variants of concern.

This paper introduces a microfluidic method to sequence HIV proviruses and their integration sites, achieving >90% capture and sequencing of paired genomes from treated patients.