Author

Jeremy A. Hengst

Pennsylvania State UniversityORCID 0000-0003-4850-1464Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Sinisa DovatiD· 2 shared Suming HuangiD· 2 shared Yasin UzuniD· 2 shared Ahsan PolashiD· 1 shared Alexis MorrisseyiD· 1 shared Arati K. SharmaiD· 1 shared Arati Sharma· 1 shared Avinash Kundadka KudvaiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPharmacology & Toxicology

Sinisa DovatORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJul 29, 2026

Oncofetal RNA-binding proteins of IGF2BP family suppress IRF-3 and NF-kB dependent transcription downstream of cytosolic RNA sensors

Garrett Knox, Ruba Agili-Shaban, Alexis Morrissey…·Irina A. Elcheva

This study shows that IGF2BP family proteins suppress RNA-sensing pathways by binding TNFAIP3 mRNA, reducing innate immune signaling in AML cells.

DOI 10.64898/2026.05.22.726816

humanbulk RNA-seq

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Pharmacology & ToxicologybioRxivPreprintDec 26, 2025

CK2 inhibitor, CX-4945, enhances BH3 priming and promotes apoptosis of venetoclax-resistant AML by targeting antiapoptotic proteins

Muhammad Daniyal, Rajesh Rajaiah, Upendarrao Golla…·Chandrika Gowda Behura

DOI 10.64898/2025.12.24.696284

humanexperimental assay

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BioinformaticsLeukemiaPeer-reviewedJun 24, 2025

Critical roles of Ikaros and HDAC1 in regulation of heterochromatin and tumor suppression in T-cell acute lymphoblastic leukemia

Yali Ding, Bing He, Daniel Bogush…·Sinisa Dovat

This study identifies Ikaros and HDAC1's roles in regulating heterochromatin and tumor suppression in T-ALL by modulating EZH2 activity and chromatin structure.

3 citations

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