Dennis Bo Jensen

This study identifies intrinsic biophysical properties of gamma motoneurons in mice, revealing mechanisms underlying their low recruitment threshold and high firing rates.

This study shows that TDP-43 pathology in the TDP43-ΔNLS mouse model drives hyperexcitability in spinal motoneurones, with reversible effects upon transgene resuppression.

The paper shows that cytoplasmic TDP-43 accumulation in a mouse model leads to changes in C-bouton number and size, with sex-specific effects on motoneurones.

The paper investigates excitability changes in G127X SOD1 mice at symptom onset, finding increased Na+ currents and longer axon initial segments (AISs) in spinal motoneurones.

This study investigates excitability of spinal motoneurones in G93A SOD1 ALS mice and finds no deficits in repetitive firing, but increased input responsiveness and persistent inward currents.