Neta Felsenthal

This study shows cancer cells follow intrinsic self-organized programs that coordinate tumor cell state transitions during growth, revealing mechanical gradients and stereotyped patterns of heterogeneity and plasticity.

This study shows that neonatal enthesis healing occurs through non-inflammatory ECM secretion by resident cells, forming an acellular scar that restores function without inflammation or angiogenesis.

The paper shows that BCKDK compensates for PDK family loss by inactivating PDC, maintaining TCA cycle function and embryonic development in mice.

The paper identifies global and regional genetic regulators that control the modular patterning of bone superstructures during long bone development.

The paper identifies the developmental transition of entheses from embryonic to mature states, showing that migrating entheses replace Sox9+ progenitors with Gli1 lineage cells.