Author

Qianyi Wu

The University of SydneyORCID 0000-0003-4991-1692Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:electronic health records· 1 Organism:human· 1

Frequent Co-Authors

Emad TajkhorshidiD· 2 shared Renae M. RyaniD· 2 shared Shashank PantiD· 2 shared Alastair Garner· 1 shared Ashkan Fakharzadeh· 1 shared Azman AkhteriD· 1 shared Chelsea Briot· 1 shared Donald J. van MeyeliD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBiophysics

Emad TajkhorshidORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

BiophysicsbioRxivPreprintNov 10, 2026

One high-order ion-coupling mechanism generates multiple SLC1A stoichiometries

Renae M Ryan, Qianyi Wu, Yan Jiang…·Emad Tajkhorshid

This study identifies a high-order ion-coupling mechanism in EAAT transporters by showing that dEAAT2 can acquire Na+, H+, and K+ dependence through mutations, mimicking human hEAAT2's complex ion coupling.

DOI 10.64898/2026.11.07.764996

Drosophilaexperimental assay

𝕏 PostBluesky

NeuroscienceJournal of Clinical InvestigationPeer-reviewedFeb 15, 2022

Ataxia-linked SLC1A3 mutations alter EAAT1 chloride channel activity and glial regulation of CNS function

Qianyi Wu, Azman Akhter, Shashank Pant…·Renae M. Ryan

The paper investigates how mutations in SLC1A3 (hEAAT1) affect chloride channel activity and glutamate transport in glial cells, linking these changes to the pathology of episodic ataxia type 6.

22 citations

humanDrosophila

𝕏 PostBluesky