Yuna Heo

This study identifies chromatin stability as critical for preserving chondrocyte function during expansion and shows that Fludarabine maintains phenotype by stabilizing chromatin and reducing STAT1 activity.

This paper develops a tunable vascular dECM platform to study stiffness-driven vascular remodeling and identifies YAP and survivin as regulators of VSMC mechanotransduction.

This study uses FLARE-ExM to examine changes in tendon cellular and extracellular structures under healthy and perturbed conditions, revealing structural alterations in response to mechanical stress.

This study shows survivin enhances VSMC migration in stiff environments, suggesting it could be a therapeutic target for arterial stiffness-related diseases.

This paper develops a tunable hydrogel system using decellularized meniscus extracellular matrix to repair zonal-specific meniscal injuries, showing improved cell differentiation and tissue integration.

This study investigates how epigenetic mechanisms regulate meniscus cell migration under inflammation, revealing zonal differences and potential therapeutic targets.