Bat‐Ider Tumenbayar

This paper develops a biomimetic scaffold system to enhance rotator cuff enthesis regeneration by replicating native tissue properties and integrating with conventional suture anchors.

This study shows ATF6α regulates lung aging and remodeling in specific cell types, with distinct effects on epithelial mitochondrial function and pericyte fibrosis.

This study shows that the Acvr1R206H mutation in FOP remodels chromatin through BMP-SMAD and Rho/ROCK signaling, driving aberrant osteogenesis, and identifies reversible chromatin changes as a therapeutic target.

This study identifies survivin as a mechanosensitive regulator linking ECM stiffness to GBM growth and matrisome remodeling, suggesting it as a therapeutic target.

This study identifies age-dependent chromatin remodeling in tendon cells that enhances inflammatory responses and contributes to tendon degeneration.

This study compares the SASP and transcriptional profiles induced by CDK4/6 inhibitors and DNA damage, revealing delayed NF-κB activation in CDK4/6i.

This study shows survivin enhances VSMC migration in stiff environments, suggesting it could be a therapeutic target for arterial stiffness-related diseases.

This study investigates how epigenetic mechanisms regulate meniscus cell migration under inflammation, revealing zonal differences and potential therapeutic targets.

This study shows that FAK and p130Cas modulate early transcription and metabolism in response to substrate stiffness using RNA sequencing in mouse cells.

The paper analyzes gene expression and protein networks in vascular injury models to show similarities with cardiovascular disease mechanisms.