Author

Felicia Pais Araújo

University of TorontoORCID 0009-0002-0675-771XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:protein sequence· 1 Assay:protein structure· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Haissi CuiiD· 1 shared Han GaoiD· 1 shared Hui PengiD· 1 shared J. P. Padilla-MartíneziD· 1 shared Jolie M. MilleriD· 1 shared Qingyu Shi· 1 shared Rasangi Tennakoon· 1 shared Samuel Protais NyandwiiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

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1 shared paperMolecular Biology

Haissi CuiORCID

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1 paper

Recent·Most cited

A disease-causing Isoleucyl-tRNA synthetase variant leads to altered protein complex formation and cellular stress response

Han Gao, Rasangi Tennakoon, Felicia Pais Araújo, Jolie M. Miller, Samuel Protais Nyandwi, Qingyu Shi, J. P. Padilla-Martínez, Hui Peng, Haissi Cui

The paper investigates a disease-causing mutation in IARS1 that affects protein complex formation and the integrated stress response, leading to altered cellular stress responses.

Journal of Biological Chemistry · Jan 23, 2026Peer-reviewedMolecular Biology