Author

Rasangi Tennakoon

University of Toronto

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Research Focus:protein design· 1 Assay:protein sequence· 1

Frequent Co-Authors

Haissi CuiiD· 3 shared Samuel Protais NyandwiiD· 3 shared Han GaoiD· 2 shared Hui PengiD· 2 shared Jolie Marie MilleriD· 2 shared Qingyu Shi· 2 shared Scott A. YuzwaiD· 2 shared Aaron R. WheeleriD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersMolecular Biology

Haissi CuiORCID

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3 papers

Recent·Most cited

A Metabolic Labeling Strategy for Tracking Protein Synthesis in Complex Biological Systems

Yong Jia Bu, Samuel Protais Nyandwi, Flavia Lima de Alves, Rasangi Tennakoon, Tabea Vanessa Stamm, David Julian Schneider, Alexander Eddenden, Timothy Wing Yin Ma, Yoo-jeong Chun, Jolie Marie Miller, Hui Peng, Aaron R. Wheeler, Scott A. Yuzwa, Mark Nitz, Haissi Cui

This paper introduces TeACAT, a method to track newly synthesized proteins in mice by incorporating a non-canonical amino acid, enabling visualization and quantification with minimal perturbation.

bioRxiv · Sep 1, 2026 · PreprintMolecular Biology

A disease-causing Isoleucyl-tRNA synthetase variant leads to altered protein complex formation and cellular stress response

Han Gao, Rasangi Tennakoon, Felicia Pais Araújo, Jolie M. Miller, Samuel Protais Nyandwi, Qingyu Shi, J. P. Padilla-Martínez, Hui Peng, Haissi Cui

The paper investigates a disease-causing mutation in IARS1 that affects protein complex formation and the integrated stress response, leading to altered cellular stress responses.

Journal of Biological Chemistry · Jan 23, 2026Peer-reviewedMolecular Biology

RARS1 integration into the multisynthetase complex is crucial for mammalian brain development

Samuel Protais Nyandwi, Rasangi Tennakoon, Qingyu Shi, Sofija Volkanoska, Danyon Harkins, Han Gao, Yong Bu, Colette Maya Macarios, Scott A. Yuzwa, Haissi Cui

This study shows that RARS1's integration into the multisynthetase complex is essential for normal brain development in mice, as its exclusion leads to developmental defects and gene dysregulation.

bioRxiv · Sep 8, 2025 · PreprintMolecular Biology