Ennis W. Deihl

This paper shows that activity-dependent mitochondrial ROS signaling in C. elegans drives avoidance plasticity by enhancing synaptic strength and circuit function.

This paper describes a protocol for using RNA interference in planarian embryos to study gene function during regeneration.

The paper identifies a new class of Dicer-dependent siRNAs in C. elegans, called 23H-RNAs, produced from diverse genomic features.

The paper shows axonal mitochondria regulate dynamic actin through reactive oxygen species, essential for electrical synapses and gentle touch response in C. elegans.

The paper demonstrates that mitochondrial ROS signaling in C. elegans regulates synaptic recruitment of glutamate receptors by modulating calcium uptake and reactive oxygen species production.