Author

Camille Gorlt

Centre National de la Recherche ScientifiqueORCID 0009-0004-0496-9449Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

Alexandre StellaiD· 1 shared Andreas MerdesiD· 1 shared Anja K. Münster-KühneliD· 1 shared Anne Gonzalez de PeredoiD· 1 shared Aurélien BoyanceiD· 1 shared Benjamin DuployeriD· 1 shared Christel VérolletiD· 1 shared Christiane BierkampiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

Alexandre StellaORCID

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2 papers

Recent·Most cited

ST8SIA4-mediated polysialylation is critical for CCR2-driven monocyte egress from the bone marrow

Aurélien Boyance, Nelly Gilles, Maxime Rotival, Alexandre Stella, Camille Gorlt, Karima Chaoui, Sarah Monard, Nacho Aguiló, Thomas Mangeat, Odile Burlet‐Schiltz, Hugues Lortat‐Jacob, Anja K. Münster-Kühnel, Anne Gonzalez de Peredo, Denis Hudrisier, Lluís Quintana‐Murci, , Yoann Rombouts

This study identifies ST8SIA4-mediated polysialylation as essential for CCR2-driven monocyte egress from the bone marrow, revealing a novel regulatory mechanism in immune cell trafficking.

bioRxiv · Jan 12, 2026 · PreprintImmunology

Loss of ninein interferes with osteoclast formation and causes premature ossification

Thierry Gilbert, Camille Gorlt, Merlin Barbier, Benjamin Duployer, Marianna Plozza, Ophélie Dufrançais, Laure-Elene Martet, Elisa Dalbard, Loelia Segot, Christophe Tenailleau, Laurence Haren, Christel Vérollet, Christiane Bierkamp, Andreas Merdes

This study shows that ninein deficiency impairs osteoclast formation and bone resorption, leading to premature ossification and altered skeletal development.

eLife · Jun 5, 2024Peer-reviewedDevelopmental Biology