Author

Jemila P. Gomes

Aarhus UniversityORCID 0009-0004-1027-8599Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Ina‐Maria RudolphiD· 2 shared Narasimha Swamy TeluguiD· 2 shared Sebastian DieckeiD· 2 shared Thomas E. WillnowiD· 2 shared A. Gręda· 1 shared Cagla CömertiD· 1 shared Duncan S. SutherlandiD· 1 shared Ewa Żurawska-PłaksejiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersNeuroscience

Ina‐Maria RudolphORCID

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2 papers

Recent·Most cited

Interaction of sortilin with apolipoprotein E3 enables neurons to use long-chain fatty acids as alternative metabolic fuel

A. Gręda, Jemila P. Gomes, Vanessa Schmidt, Ewa Żurawska-Płaksej, Raphaela Fritsche‐Guenther, Ina‐Maria Rudolph, Narasimha Swamy Telugu, Cagla Cömert, Jennifer Kirwan, Séverine Kunz, Michael Rothe, Mogens Johannsen, Sebastian Diecke, Peter Bross, Thomas E. Willnow

The paper shows sortilin mediates neuronal uptake of fatty acids via apoE, enabling their use as metabolic fuel, but this pathway is disrupted by apoE4, linked to Alzheimer's risk.

Nature Metabolism · Oct 16, 2025Peer-reviewedNeuroscience

Familial Alzheimer disease mutation identifies novel role of SORLA in release of neurotrophic exosomes

Kristian Juul‐Madsen, Ina‐Maria Rudolph, Jemila P. Gomes, Katrina Meyer, Peter Lund Ovesen, Malgorzata J. Górniak-Walas, Marianna Kokoli, Narasimha Swamy Telugu, Malthe von Tangen Sivertsen, Duncan S. Sutherland, Johan Palmfeldt, Sebastian Diecke, Olav M. Andersen, Matthias Selbach, Thomas E. Willnow

This study identifies a novel role for SORLA in regulating exosome release and neurotrophic function, linking SORL1 mutations to Alzheimer's disease pathology.

bioRxiv · Apr 22, 2025 · PreprintNeuroscience