Caroline Dillard

This study identifies cooperative and suppressive interclonal interactions during early Ras-driven tumourigenesis using a Drosophila model, revealing key modifiers like SWI/SNF and cell competition regulators.

This study shows that oncogenic RasV12 activates Dorsal/NF-κB signaling, promoting tumor growth and malignancy in Drosophila melanogaster tumors.

The paper introduces EyaHOST, a genetic system in Drosophila for studying tumor-host interactions by enabling tissue-specific manipulation of host cells and tissues.

The paper shows that chinmo is silenced in Drosophila neuroblasts and neuroepithelial cells through different mechanisms to regulate self-renewal.