Author

Masaya Inoko

Hokkaido UniversityORCID 0009-0005-0286-6435Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Research Focus:development & morphogenesis· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:imaging· 1

Frequent Co-Authors

Guang YangiD· 3 shared Ryota UeharaiD· 3 shared Yuki TsukadaiD· 3 shared Akira FunahashiiD· 1 shared Kaito Ogura· 1 shared Masanao SatoiD· 1 shared Sumire Ishida‐IshiharaiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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3 shared papersCell Biology

Guang YangORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintAug 26, 2026

Centrosome-centromere capture range, rather than centrosome arrangement, determines multipolar chromosome segregation pattern after whole-genome duplication

Masaya Inoko, Guang Yang, Yuki Tsukada, Ryota Uehara

DOI 10.64898/2026.08.24.746909

experimental assaychromatin & epigenetic regulation

𝕏 PostBluesky

Cell BiologyBiology OpenPeer-reviewedMay 11, 2026

Profiling cell proliferation after whole-genome duplication in human cells

Guang Yang, Masaya Inoko, Kaito Ogura…·Ryota Uehara

The paper uses live-imaging to study proliferation dynamics of post-WGD HCT116 cell lineages, finding that multipolar chromosome segregation limits proliferative capacity.

2 citationstop 4%

𝕏 PostBluesky

Cell BiologyProceedings of the National Academy of SciencesPeer-reviewedApr 15, 2026

Sister chromatid separation determines the proliferative properties upon whole-genome duplication via homologous chromosome arrangement

Masaya Inoko, Guang Yang, Yuki Tsukada, Ryota Uehara

This study shows that mitotic slippage and cytokinesis failure, two mechanisms of whole-genome duplication, differentially affect post-WGD cell viability through distinct chromosome segregation patterns.

2 citationstop 4%

humanproteomics

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