Author

Verena Bopp

University and Rehabilitation Clinics UlmORCID 0009-0006-1928-5052Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 4 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 4 Approach:experimental assay· 4 Organism:mouse· 4 Approach:statistical model· 4 Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Assay:snRNA-seq· 2 Assay:CITE-seq· 1

Frequent Co-Authors

Karin M. DanzeriD· 5 shared Veselin GrozdanoviD· 4 shared Jaehyun LeeBaeiD· 3 shared Julia K. Kühlwein· 3 shared Benjamin MayeriD· 2 shared Daniel Rombach· 2 shared Hartmut GeigeriD· 2 shared Laura Meier· 2 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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5 shared papersNeuroscience

Karin M. DanzerORCID

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5 papers

Recent·Most cited

NeurosciencebioRxivPreprintSep 1, 2026

Dysregulated splenic glucocorticoid sensitivity in aging and an α-synuclein transgenic mouse model of Parkinson's disease

Daniel Rombach, Verena Bopp, Dominik Langgartner…·Karin M. Danzer

This study investigates how aging and α-synuclein pathology affect glucocorticoid sensitivity in splenocytes, finding reduced sensitivity in aged mice and PD models at specific ages.

DOI 10.64898/2026.08.27.745197

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 14, 2026

Targeting CDC42 with CASIN Reprograms Cell-Type-Specific Transcriptomes and MAPK-Driven Transcription Factor Networks in the Aging Brain

Jaehyun LeeBae, Verena Bopp, Bettina Moehrle…·Karin M. Danzer

The paper shows that CASIN inhibits CDC42, reversing PD-related transcriptional changes and affecting MAPK and PI3K/AKT pathways in an aging mouse model.

DOI 10.64898/2026.08.07.742974

mousesnRNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 27, 2025

Molecular aging is the main driver of Parkinson's Disease

Verena Bopp, Jaehyun LeeBae, Patrick Oeckl…·Karin M. Danzer

This study shows that aging processes drive Parkinson's Disease in a mouse model, independent of alpha-synuclein oligomers, by targeting CDC42 activity.

DOI 10.1101/2025.08.27.672359

mousescRNA-seq

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GenomicsbioRxivPreprintAug 12, 2025

Monocyte Dysregulation Defines an MDD-Specific Transcriptional Signature Closely Linked to Clinical MDD Traits

Laura Meier, Medhanie Assmelash Mulaw, Verena Durner…·Karin M. Danzer

The paper identifies a monocyte dysregulation signature in MDD linked to clinical symptoms using CITE-Seq data from PBMCs.

DOI 10.1101/2025.08.12.669833

humanCITE-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 8, 2025

Spreading alpha-Synuclein Oligomers Trigger Astrocyte Changes and Astrocyte-Glutamatergic Neuron system dysfunction in an Age-related Manner

Jaehyun LeeBae, Verena Bopp, Alicia Goreth…·Karin M. Danzer

The paper investigates how alpha-synuclein oligomers spread in a mouse model of Parkinson's disease, identifying age-related astrocyte changes and dysfunction in the astrocyte-glutamatergic neuron system.

DOI 10.1101/2025.08.08.669249

mousespatial transcriptomics

𝕏 PostBluesky