Karin Schön

This paper presents a synthetic virology platform for rapidly prototyping influenza A virus mutants with altered antigenic sites using computational design and artificial synthesis.

This study shows nasal tissue-resident memory CD4+ T cells persist after influenza infection and offer protection against different virus strains.

This study investigates how vaccine antigen valency and pre-existing memory B cells influence B cell responses to drifted influenza virus, showing differential effects on germinal center dynamics and immunodominance.

This study shows that influenza A virus exploits nuclear RNAi by inducing AGO2 accumulation and silencing type-I interferon genes, and that arsenic trioxide can counteract this effect.

This study designs stem-epitope mimetics using computational methods to elicit broadly reactive antibodies against influenza viruses.

The paper identifies a nanobody that targets a conserved epitope on influenza A virus H7 hemagglutinin, providing cross-subtype neutralization and protection.