Author

A. Parini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Henras· 1 shared A. Lagnado· 1 shared A. Walaszczyk· 1 shared A.-C. Godet· 1 shared A.-C. Prats· 1 shared B. Bournet· 1 shared B. Garmy-Susini· 1 shared B. Guiard· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCell Biology

A. Henras

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pathogenicity of Commensal Gut Biofilm In Prefrail Aging

G. Le Cosquer, M. Pannier, E. S. Meunier, J. Thevenin, E. Pyhourquet, S. Guyonnet, B. Vellas, Y. Santin, B. Guiard, A. Parini, L. Buscail, B. Bournet, D. Guillemet, C. Deraison, N. Vergnolle, J. P. Motta

The paper investigates prefrail gut microbiota biofilm changes and their inflammatory effects, showing potential for microbiota-targeted interventions in aging.

bioRxiv · Jun 4, 2024Microbiology

Vasohibin1, a new IRES trans-acting factor for induction of (lymph)angiogenic factors in early hypoxia

F. Hantelys, A.-C. Godet, F. David, F. Tatin, E. Renaud-Gabardos, F. Pujol, L. Diallo, I. Ader, L. Ligat, A. Henras, Y. Sato, E. Lacazette, A. Parini, B. Garmy-Susini, A.-C. Prats

The paper identifies vasohibin1 as an IRES trans-acting factor that activates angiogenic IRESs in early hypoxia while inhibiting others in normoxia.

bioRxiv · Jul 11, 2019 · PreprintCell Biology

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani, A. Walaszczyk, E. Dookun, J. Chapman, J. Birch, H. Salmonowicz, M. Ogrodnik, D. Jurk, C. Proctor, C. Correia-Melo, S. Victorelli, E. Fielder, R. Berlinguer-Palmini, , J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

bioRxiv · Aug 17, 2018Cell Biology