Author

Allyson Cole-Strauss

Michigan State University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:drug response / drug discovery· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:imaging· 1 Organism:non-human primate· 1 Assay:proteomics· 1

Frequent Co-Authors

Caryl E. SortwelliD· 3 shared Kelvin C. LukiD· 3 shared Christopher J. Kemp· 2 shared Jack W. LiptoniD· 2 shared Joseph R. PattersoniD· 2 shared Kathy Steece‐CollieriD· 2 shared Nathan C. Kuhn· 2 shared Alison I. BernsteiniD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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3 shared papersNeuroscience

Caryl E. SortwellORCID

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Author profile →

3 papers

Recent·Most cited

Complement Dysregulation During the Early Phases of Synucleinopathy

Hina Khan, Mary Gifford, Arash Kordbacheh, Asher Bury, Spencer Panoushek, Allyson Cole-Strauss, Christopher J. Kemp, Kelvin C. Luk, Kathy Steece‐Collier, Nathan C. Kuhn, Nicholas M. Kanaan, Caryl E. Sortwell, Joseph R. Patterson, Matthew J. Benskey

This study shows that synucleinopathy triggers complement activation and dysregulation before neurodegeneration in a rat model and human Parkinson's disease tissue.

Acta Neuropathologica · Jul 20, 2026Peer-reviewedNeuroscience

Developmental exposure to the organochlorine pesticide dieldrin causes male-specific exacerbation of α-synuclein-preformed fibril-induced toxicity and motor deficits

Aysegul O. Gezer, Joseph J. Kochmanski, Sarah VanOeveren, Allyson Cole-Strauss, Christopher J. Kemp, Joseph R. Patterson, Kathryn M. Miller, Nathan C. Kuhn, Danielle E. Herman, Alyssa McIntire, Jack W. Lipton, Kelvin C. Luk, Sheila M. Fleming, Caryl E. Sortwell, Alison I. Bernstein

This study shows that developmental dieldrin exposure increases male-specific motor deficits and dopamine handling issues in a synucleinopathy model, without affecting nigral neuron loss.

Neurobiology of Disease · May 15, 2020Peer-reviewedNeuroscience

Alpha-synuclein antisense oligonucleotides as a disease-modifying therapy for Parkinson’s disease

Tracy A. Cole, Hien Tran Zhao, Timothy J. Collier, Ivette M. Sandoval, Caryl E. Sortwell, Kathy Steece‐Collier, Brian F. Daley, Alix Booms, Jack W. Lipton, Mackenzie Welch, Melissa Berman, Luke Jandreski, Danielle L. Graham, Andreas Weihofen, Stephanie L. Celano, , Katrina L. Paumier

This paper demonstrates that antisense oligonucleotides targeting SNCA reduce alpha-synuclein production, prevent pathology, and show translational potential in non-human primates.

bioRxiv · Nov 4, 2019 · PreprintNeuroscience