Author

Alvin Huang

Harvard UniversityPrimary ORCID profile (0009-0000-9476-8675 · 5 papers)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Alvin HuangiD5 papers · Allen Institute for Brain Science

Biology Disciplines

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Microbiology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Allison Rose HanafordiD· 1 shared Anastasia Dimitriou· 1 shared Ashley Ching· 1 shared Benjamin E. GewurziD· 1 shared Bo ZhaoiD· 1 shared Brittany M. JohnsoniD· 1 shared Chong WangiD· 1 shared David JS Zhao· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Allison Rose HanafordORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A Human Genome-wide CRISPR-Cas9 Screen Reveals Host Factors That Support Epstein-Barr Virus B cell infection

Hongbo Wang, Yao Yu Yeo, Chong Wang, Rui Guo, Shaowen White, Yifei Liao, Stephanie Yiu, Gloria Liu, Alvin Huang, David JS Zhao, Zongwei Wang, Sizun Jiang, Benjamin E. Gewurz, Bo Zhao

This study uses a genome-wide CRISPR-Cas9 screen to identify host factors essential for EBV B cell infection, revealing new pathways and potential therapeutic targets.

bioRxiv · Sep 28, 2026 · PreprintMicrobiology

MyD88 deficiency modestly attenuates disease in a Leigh syndrome mouse model while enrofloxacin accelerates disease

Allison Rose Hanaford, Elizaveta A. Olkhova, Ryan W. Liao, Ashley Ching, Alvin Huang, Erin Shien Hsieh, Kino Watanabe, Yihan Chen, Megan Wichman, Noelle Hwang, Katerina James, Michael T. Mulholland, Vivian Truong, Holly Coulson, Kerry Gibbins, , Simon C. Johnson

The paper investigates the role of TLR signaling in Leigh syndrome using a mouse model and finds that MyD88 deficiency modestly improves disease outcomes, while enrofloxacin treatment accelerates disease progression.

Journal of Neuropathology & Experimental Neurology · Sep 4, 2026Peer-reviewedNeuroscience