Author

Amanda B. Nymon

Dartmouth College

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Bruce A. StantoniD· 3 shared Roxanna Barnaby· 3 shared Carolyn Roche· 2 shared Lily A. CharpentieriD· 2 shared Thomas H. HamptoniD· 2 shared Benjamin D. RossiD· 1 shared Byoung-Kyu ChoiD· 1 shared Daniel J. WeissiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersMicrobiology

Bruce A. StantonORCID

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3 papers

Recent·Most cited

Outer Membrane Vesicles Secreted by Bacteroides fragilis Inhibit CFTR Chloride Secretion by Human Colon Organoids

Roxanna Barnaby, Amanda B. Nymon, Paige Salerno, Carolyn Roche, Young Ah Goo, Byoung-Kyu Cho, Timothy B. Gardner, Zdeněk Švindrych, Douglas J. Taatjes, Thomas H. Hampton, Benjamin D. Ross, Bruce A. Stanton

This study shows that Bacteroides fragilis outer membrane vesicles reduce CFTR Cl- secretion in human colon organoids, with BFT-2-containing vesicles being more effective.

Infection and Immunity · May 29, 2026Peer-reviewedMicrobiology

Let-7b-5p loaded Mesenchymal Stromal Cell Extracellular Vesicles reduce Pseudomonas- biofilm formation and inflammation in CF Bronchial Epithelial Cells

Sharanya Sarkar, Roxanna Barnaby, Amanda B. Nymon, Lily A. Charpentier, Lily D. Taub, Matthew J. Wargo, Daniel J. Weiss, Tracey L. Bonfield, Bruce A. Stanton

This paper shows that let-7b-5p-loaded MSC EVs block antibiotic-resistant P. aeruginosa biofilm formation and reduce inflammation in CF bronchial epithelial cells.

bioRxiv · May 28, 2025 · PreprintMicrobiology

Gene expression responses of CF airway epithelial cells exposed to elexacaftor/tezacaftor/ivacaftor (ETI) suggest benefits beyond improved CFTR channel function

Thomas H. Hampton, Roxanna Barnaby, Carolyn Roche, Amanda B. Nymon, Kiyoshi F. Fukutani, Todd A. MacKenzie, Lily A. Charpentier, Bruce A. Stanton

This study shows ETI increases defensin gene expression and reduces MMP10, MMP12, and inflammatory signaling in CF airway epithelial cells.

American Journal of Physiology-Lung Cellular and Molecular Physiology · Oct 22, 2024Peer-reviewedPharmacology & Toxicology