Author

B. Aschenbrenner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 3 Organism:human· 3 Assay:scRNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

B. M. Lichtenberger· 2 shared J. Griss· 2 shared V. Nguyen· 2 shared W. Weninger· 2 shared A. Abdollahi· 1 shared A. Dubrovska· 1 shared A. Forsthuber· 1 shared A. Franken· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCancer Biology

B. M. Lichtenberger

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3 papers

Recent·Most cited

Cancer associated fibroblast subtypes modulate the tumor-immune microenvironment and are associated with skin cancer malignancy

A. Forsthuber, B. Aschenbrenner, A. Korosec, T. Jacob, K. Annusver, N. Krajic, D. Kholodniuk, S. Frech, S. Zhu, K. Purkhauser, K. Lipp, F. Werner, V. Nguyen, J. Griss, W. Bauer, , B. M. Lichtenberger

This study identifies three CAF subtypes in skin cancers and shows their association with tumor malignancy and immune modulation.

bioRxiv · May 3, 2023Cancer Biology

Indolent primary cutaneous B-cell lymphomas resemble persistent antigen reactions without signs of dedifferentiation

J. Griss, S. Gansberger, I. Oyarzun, M. C. Drach, V. Nguyen, L. E. Shaw, U. Mann, S. Porkert, M. Farlik, W. Weninger, W. Dolak, B. Aschenbrenner, B. M. Lichtenberger, C. Wagner, I. Simonitsch-Klupp, , P. M. Brunner

This study uses single-cell RNA sequencing to compare indolent primary cutaneous B-cell lymphomas with other B-cell proliferations, finding they resemble persistent antigen reactions with germinal center features.

bioRxiv · Dec 16, 2022 · PreprintCancer Biology

GLS-driven glutamine catabolism contributes to prostate cancer radiosensitivity by regulating the redox state, stemness and ATG5-mediated autophagy

A. Mukha, U. Kahya, A. Linge, O. Chen, S. Lock, V. Lukiyanchuk, S. Richter, T. C. Alves, M. Peitzsch, V. Telychko, S. Skvortsov, G. Negro, B. Aschenbrenner, I.-I. Skvortsova, P. Mirtschink, , A. Dubrovska

This study shows that glutamine catabolism via GLS regulates redox state, stemness, and autophagy in prostate cancer, affecting radiosensitivity and patient outcomes.

bioRxiv · Jan 7, 2021Cancer Biology