Fibroblasts from metastatic sites induce broad-spectrum drug desensitization via modulation of mitochondrial priming
B. D. Landry, T. Leete, R. Richards, P. Cruz-Gordillo, G. Ren, A. D. Schwartz, S. R. Peyton, M. J. Lee
The paper identifies how fibroblasts from metastatic sites modulate mitochondrial priming in tumor cells, leading to drug desensitization through inflammatory cytokine secretion.
bioRxiv · Oct 2, 2017Cancer Biology