Author

T. Leete

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

M. J. Lee· 2 shared P. Cruz-Gordillo· 2 shared A. D. Schwartz· 1 shared B. D. Landry· 1 shared G. Ren· 1 shared M. E. Honeywell· 1 shared R. Richards· 1 shared S. R. Peyton· 1 shared

Activity by Year

2017-2020 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 23 mutual co-author links

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2 shared papersCancer Biology

M. J. Lee

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2 papers

Recent·Most cited

ELP-dependent expression of MCL1 promotes resistance to EGFR inhibition in triple-negative breast cancer cells

P. Cruz-Gordillo, M. E. Honeywell, T. Leete, M. J. Lee

The study identifies the ELP complex as a mediator of resistance to EGFR inhibition in TNBC by promoting MCL1 expression, suggesting co-targeting EGFR and MCL1 as a therapeutic strategy.

bioRxiv · Mar 29, 2020Cancer Biology

Fibroblasts from metastatic sites induce broad-spectrum drug desensitization via modulation of mitochondrial priming

B. D. Landry, T. Leete, R. Richards, P. Cruz-Gordillo, G. Ren, A. D. Schwartz, S. R. Peyton, M. J. Lee

The paper identifies how fibroblasts from metastatic sites modulate mitochondrial priming in tumor cells, leading to drug desensitization through inflammatory cytokine secretion.

bioRxiv · Oct 2, 2017Cancer Biology