Author

R. Richards

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1 Ecology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1

Frequent Co-Authors

A. D. Schwartz· 1 shared B. D. Landry· 1 shared D. Mason· 1 shared G. Ren· 1 shared H. Mokhtarzadeh· 1 shared J. Drake· 1 shared M. J. Lee· 1 shared P. Cruz-Gordillo· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. D. Schwartz

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Self-paced treadmill controller algorithm based on position and speed of centre of mass

H. Mokhtarzadeh, R. Richards, T. Geijtenbeek

The paper introduces a self-paced treadmill controller algorithm using center of mass position and velocity to adjust belt speed, validated with over 410 subjects and simulated with different sensitivities.

bioRxiv · Jun 21, 2022 · PreprintBioengineering

4D imaging and analysis of multicellular tumour spheroid cell migration and invasion

R. Richards, D. Mason, R. Kelly, R. Levy, R. Bearon, V. See

This study uses 4D imaging to analyze 3D cell migration and invasion in glioblastoma spheroids, evaluating chemotherapy efficacy on invasion.

bioRxiv · Sep 3, 2019 · PreprintCell Biology

Estimating environmental suitability

J. Drake, R. Richards

The paper defines environmental suitability as the conditional probability of species occurrence and introduces a new method for species distribution modeling.

Ecosphere · Sep 1, 2018Peer-reviewedEcology

Fibroblasts from metastatic sites induce broad-spectrum drug desensitization via modulation of mitochondrial priming

B. D. Landry, T. Leete, R. Richards, P. Cruz-Gordillo, G. Ren, A. D. Schwartz, S. R. Peyton, M. J. Lee

The paper identifies how fibroblasts from metastatic sites modulate mitochondrial priming in tumor cells, leading to drug desensitization through inflammatory cytokine secretion.

bioRxiv · Oct 2, 2017Cancer Biology