Author

A. D. Schwartz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:drug response / drug discovery· 2 Assay:bulk RNA-seq· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

L. E. Barney· 3 shared C. L. Hall· 2 shared L. E. Jansen· 2 shared S. Peyton· 2 shared S. R. Peyton· 2 shared A. S. Meyer· 1 shared B. D. Landry· 1 shared C. C. Babbitt· 1 shared

Activity by Year

2017-2018 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersBioengineering

L. E. Barney

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Integrin α6 and EGFR Signaling Converge at Mechanosensitive Calpain 2

A. D. Schwartz, C. L. Hall, L. E. Barney, C. C. Babbitt, S. R. Peyton

The paper investigates how integrin α6 and EGFR signaling converge through mechanosensitive calpain 2 in carcinoma progression.

bioRxiv · Apr 15, 2018Bioengineering

Anomalous Diffusion as a Descriptive Model of Cell Migration

I. D. Luzhanskey, J. P. MacMunn, J. D. Cohen, L. E. Barney, L. E. Jansen, A. D. Schwartz, S. Peyton

This study shows that an anomalous diffusion model better describes cell migration in 2D and 3D environments than the persistent random walk model, especially in subdiffusive conditions.

bioRxiv · Dec 18, 2017Bioengineering

A Biomaterial Screening Approach to Reveal Microenvironmental Mechanisms of Drug Resistance

A. D. Schwartz, L. E. Barney, L. E. Jansen, T. V. Nguyen, C. L. Hall, A. S. Meyer, S. Peyton

This paper screens drug responses across biomaterial platforms to identify ECM-mediated mechanisms of resistance to RTK-targeted therapies and finds that MEK phosphorylation is a key factor.

bioRxiv · Nov 20, 2017Bioengineering

Fibroblasts from metastatic sites induce broad-spectrum drug desensitization via modulation of mitochondrial priming

B. D. Landry, T. Leete, R. Richards, P. Cruz-Gordillo, G. Ren, A. D. Schwartz, S. R. Peyton, M. J. Lee

The paper identifies how fibroblasts from metastatic sites modulate mitochondrial priming in tumor cells, leading to drug desensitization through inflammatory cytokine secretion.

bioRxiv · Oct 2, 2017Cancer Biology