Author

B. M. Kuemmerer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. vom Hemdt· 1 shared C. Gruetzner· 1 shared D. J. Lapp· 1 shared E. Bartok· 1 shared E. Latz· 1 shared F. I. Schmidt· 1 shared F. N. Gohr· 1 shared G. Hartmann· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. vom Hemdt

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2 papers

Recent·Most cited

P38 kinases mediate NLRP1 inflammasome activation after ribotoxic stress response and virus infection

L.-M. Jenster, K.-E. Lange, S. Normann, A. vom Hemdt, J. D. Wuerth, L. D. J. Schiffelers, Y. M. Tesfamariam, F. N. Gohr, L. Klein, I. H. Kaltheuner, D. J. Lapp, J. Mayer, J. Moecking, H. L. Ploegh, E. Latz, , F. I. Schmidt

This study identifies p38 kinases as a central signaling hub for NLRP1 inflammasome activation in response to ribotoxic stress and virus infection.

bioRxiv · Jan 24, 2022Immunology

RIG-I-induced innate antiviral immunity protects mice from lethal SARS-CoV-2 infection

S. Marx, B. M. Kuemmerer, C. Gruetzner, H. Kato, M. Schlee, E. Bartok, M. Renn, G. Hartmann

This study shows that activating the RIG-I receptor with synthetic RNA oligonucleotides before SARS-CoV-2 infection improves mouse survival and reduces viral load.

bioRxiv · Aug 6, 2021Immunology