Author

S. Normann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:statistical model· 1

Frequent Co-Authors

F. I. Schmidt· 2 shared H. L. Ploegh· 2 shared L. D. J. Schiffelers· 2 shared M. Geyer· 2 shared A. Alon· 1 shared A. Kopp· 1 shared A. vom Hemdt· 1 shared B. M. Kuemmerer· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersImmunology

F. I. Schmidt

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Antagonistic nanobodies reveal mechanism of GSDMD pore formation and unexpected therapeutic potential

L. D. J. Schiffelers, S. Normann, S. C. Binder, E. Hagelauer, A. Kopp, A. Alon, M. Geyer, H. L. Ploegh, F. I. Schmidt

This study uses nanobodies to reveal GSDMD pore formation mechanism and shows their therapeutic potential in preventing pyroptosis.

bioRxiv · Apr 20, 2023Immunology

P38 kinases mediate NLRP1 inflammasome activation after ribotoxic stress response and virus infection

L.-M. Jenster, K.-E. Lange, S. Normann, A. vom Hemdt, J. D. Wuerth, L. D. J. Schiffelers, Y. M. Tesfamariam, F. N. Gohr, L. Klein, I. H. Kaltheuner, D. J. Lapp, J. Mayer, J. Moecking, H. L. Ploegh, E. Latz, , F. I. Schmidt

This study identifies p38 kinases as a central signaling hub for NLRP1 inflammasome activation in response to ribotoxic stress and virus infection.

bioRxiv · Jan 24, 2022Immunology