Author

B. von Eyss

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Marszalek· 1 shared A. Sanz-Moreno· 1 shared C. Guenes· 1 shared C. Kaether· 1 shared C. P. Ade· 1 shared D. Gertzmann· 1 shared F. Liss· 1 shared F. Zenk· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCell Biology

A. Marszalek

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A YIPF5-GOT1A/B complex directs a transcription independent function of ATF6 in ER export

P. Cramer, Y. Yonemura, L. Behrendt, A. Marszalek, M. Sannai, W. Durso, C. Guenes, K. Szafranski, N. Nakamura, T. Nasrashvili, J. Mayer, B. von Eyss, C. Kaether

The paper identifies YIPF5 and GOT1A/B as components of ER export and shows ATF6 has a transcription-independent role in ER export inhibition by FLI-06.

bioRxiv · Dec 12, 2023 · PreprintCell Biology

TRPS1 maintains luminal progenitors in the mammary gland by repressing SRF/MRTF activity

M. Tollot-Wegner, M. A. Jessen, K. Kim, A. Sanz-Moreno, N. Spielmann, V. Gailus-Durner, H. Fuchs, M. Hrabe de Angelis, B. von Eyss

This study shows TRPS1 maintains luminal progenitors in the mammary gland by repressing SRF/MRTF activity, linking it to mechanotransduction and breast cancer initiation.

bioRxiv · Nov 24, 2023Cell Biology

Inhibition of the YAP-MMB interaction and targeting NEK2 as potential therapeutic strategies for YAP-driven cancers

M. Jessen, D. Gertzmann, F. Liss, F. Zenk, L. Baehner, V. Schoeffler, C. P. Ade, B. von Eyss, S. Gaubatz

This study identifies disrupting YAP-B-MYB interaction and targeting NEK2 as potential therapies for YAP-driven cancers, showing reduced tumorigenesis and cell cycle defects.

bioRxiv · Jul 27, 2023Cancer Biology