Author

Brie Beck

University at Buffalo, State University of New York

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:electronic health records· 1

Frequent Co-Authors

Amanda H. KleiniD· 2 shared Kayla JohnsoniD· 2 shared Belle Meyer· 1 shared Bethany Theresa Nelson· 1 shared Cassidy Hill· 1 shared Christian Kriner· 1 shared Clémence Roué· 1 shared Ethan McFerland· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Amanda H. KleinORCID

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3 papers

Recent·Most cited

Loss of SUR1 and Kir6.2 ATP-sensitive potassium channel subunits produces comparable neuronal excitability and morphine antinociception

Soomin C. Song, Brie Beck, Kayla Johnson, Sunna Mujteba, William A. Coetzee, Amanda H. Klein

This study shows that loss of either SUR1 or Kir6.2 subunits in neurons increases dorsal root ganglion excitability and reduces morphine antinociception in mice.

bioRxiv · Oct 28, 2026 · PreprintNeuroscience

FABP8/PMP2 is a positive regulator of PNS myelination

Jiayue Hong, Clémence Roué, Simar Arora, Ethan McFerland, Sophia Elston, Khushi N. Panchal, Olivia Harris, Mathieu R. DiPersio, Taylor Humphrey, Brie Beck, Kaitlin Munro, Senbahavalli Ramasamy, Yannick Poitelon, Sophie Belin

This study shows that increasing PMP2 in Schwann cells enhances myelin thickness without affecting ultrastructure or nerve function.

bioRxiv · Jun 29, 2026 · PreprintNeuroscience

Inhibition of adenylyl cyclase 1 (AC1) and exchange protein directly activated by cAMP (EPAC) restores ATP-sensitive potassium (KATP) channel activity after chronic opioid exposure

Amanda H. Klein, Sabbir Alam, Kayla Johnson, Christian Kriner, Brie Beck, Bethany Theresa Nelson, Cassidy Hill, Belle Meyer, Jonas Mellang, Val J. Watts

This study shows that inhibiting AC1 and EPAC restores KATP channel activity after chronic opioid exposure, reducing morphine tolerance and withdrawal.

bioRxiv · Feb 3, 2025 · PreprintNeuroscience