Author

C. D. Goodman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 2 Organism:plant (other)· 2 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

G. I. McFadden· 2 shared A. E. Barry· 1 shared A. F. Cowman· 1 shared B. E. Sleebs· 1 shared B. Kouskousis· 1 shared B. S. Crabb· 1 shared C. Boulet· 1 shared C. Evelyn· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

G. I. McFadden

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2 papers

Recent·Most cited

Aryl amino acetamides prevent the development of Plasmodium falciparum rings via inhibition of the lipid transfer protein PfSTART1

M. G. Dans, C. Boulet, G. M. Watson, W. Nguyen, J. M. Dziekan, C. Evelyn, K. Reaksudsan, S. Mehra, Z. Razook, N. D. Geoghegan, M. J. Mlodzianoski, C. D. Goodman, D. B. Ling, T. K. Jonsdottir, J. Tong, , P. R. Gilson

The paper identifies aryl amino acetamides that inhibit Plasmodium falciparum ring development by targeting PfSTART1, a lipid transfer protein, and shows these inhibitors block parasite transmission.

bioRxiv · Nov 2, 2023 · PreprintCell Biology

A single point mutation in the Plasmodium falciparum ftsh1 metalloprotease confers actinonin resistance.

C. D. Goodman, T. Uddin, N. J. Spillman, G. I. McFadden

This study identifies a single point mutation in PfFtsH1 as the cause of actinonin resistance in P. falciparum, confirming PfFtsH1 as the drug's target.

eLife · Jul 17, 2020Peer-reviewedMicrobiology