Author

C. Huesa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

C. Farquharson· 2 shared A. R. Guntur· 1 shared A. S. Werber· 1 shared B. J. Thomas· 1 shared C. J. Rosen· 1 shared C. Vary· 1 shared D. Ball· 1 shared G. Karsenty· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersPathology

C. Farquharson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Developing and Investigating a Nanovibration Intervention for the Prevention/Reversal of Bone Loss Following Spinal Cord Injury

J. A. Williams, P. Campsie, R. Gibson, O. Johnson-Love, A. S. Werber, M. Sprott, R. Meechan, C. Huesa, J. F. C. Windmill, M. Purcell, S. Coupaud, M. J. Dalby, P. G. Childs, J. S. Riddell, S. Reid

This study tests a nanovibration device for reversing bone loss after spinal cord injury in rats, finding no reversal but increased collagen synthesis markers.

bioRxiv · Feb 12, 2024Bioengineering

The role of accelerated growth plate fusion in the absence of SOCS2 on osteoarthritis vulnerability

H. J. Samvelyan, C. Huesa, L. Cui Lin, C. Farquharson, K. A. Staines

This study examines whether accelerated growth due to SOCS2 deletion affects osteoarthritis vulnerability in mice, finding no effect on disease susceptibility despite changes in growth plate fusion.

bioRxiv · May 13, 2021Pathology

PHOSPHO1, a novel skeletal regulator of insulin resistance and obesity

K. J. Suchacki, N. M. Morton, C. Vary, C. Huesa, M. C. Yadav, B. J. Thomas, S. Rajoanah, L. Bunger, D. Ball, M. Barrios-Llerena, A. R. Guntur, Z. Khavandgar, W. P. Cawthorn, M. Ferron, G. Karsenty, , C. Farquharson

This study shows that PHOSPHO1 deficiency improves glucose homeostasis and protects against obesity and diabetes without altering osteocalcin levels.

bioRxiv · May 4, 2020 · PreprintPhysiology