Author

C. Reeves

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. Blackford· 1 shared A. Eisenberg· 1 shared A. Elbourne· 1 shared A. Gelmi· 1 shared A. Henssen· 1 shared A. Kentsis· 1 shared A.-K. Emde· 1 shared C. Antonescu· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCancer Biology

A. Blackford

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Real-time Biomechanical Characterisation of Cytoskeletal Remodelling

K. Zhang, C. Reeves, J. D. Berry, K. Fox, A. Elbourne, A. Gelmi

This study uses Q-AFM to track real-time biomechanical changes in hMSCs during cytoskeletal remodelling induced by disruptors.

bioRxiv · May 29, 2024 · PreprintBiophysics

Amikacin potentiator activity of zinc complexed to a pyrithione derivative with enhanced solubility

J. Magallon, P. Vu, C. Reeves, S. Kwan, K. Phan, C. L. Oakley-Havens, K. Rocha, V. Jimenez, M. S. Ramirez, M. E. Tolmasky

This paper tests a zinc-pyrithione derivative with improved solubility as an amikacin potentiator against antibiotic-resistant bacteria.

bioRxiv · Oct 8, 2021Microbiology

Human PGBD5 DNA transposase promotes site-specific oncogenic mutations in rhabdoid tumors

A. Henssen, R. Koche, J. Zhuang, E. Jiang, C. Reed, A. Eisenberg, E. Still, I. MacArthur, E. Rodriguez-Fos, S. Gonzalez, M. Puiggros, A. Blackford, C. Mason, E. De Stanchina, M. Gonen, , A. Kentsis

This study identifies PGBD5 as an oncogenic DNA transposase in rhabdoid tumors, causing site-specific genomic rearrangements through PSS sequences and end-joining repair.

bioRxiv · Feb 23, 2017Cancer Biology