Author

COSIPOP study group

Institut Pasteur

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anne DerbiseiD· 1 shared Antoine Thomas· 1 shared Arnaud MengiD· 1 shared Carmen BuchrieseriD· 1 shared Caroline BuscailiD· 1 shared Catherine WertsiD· 1 shared Christian E. DemeureiD· 1 shared Cyril SaviniD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperMicrobiology

Anne DerbiseORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

β-Lactam antibiotics trigger mouse TLR2/4- and human TLR2/5-dependent Jarisch-Herxheimer reaction in leptospirosis

Stylianos Papadopoulos, Thomas Bernard, Dorian Joffres, Frédérique Vernel-Pauillac, Julie Cagliero, COSIPOP study group, Catherine Werts

This study shows beta-lactam antibiotics induce a Jarisch-Herxheimer reaction in leptospirosis through TLR2/4 and TLR2/5-dependent mechanisms in mice and humans.

bioRxiv · Sep 10, 2026 · PreprintImmunology

Single-cell metabolomics reveals infection-specific metabolic reprogramming of human macrophages

Paula Martínez-Oca, Arnaud Meng, Quentin Vanbellingen, Marvin Albert, Antoine Thomas, Perrine Bomme, Francisco Javier García-Rodríguez, COSIPOP study group, Jean-Yves Tinévez, Sandrine Aros-Calt, Carmen Buchrieser, Pedro Escoll

This study uses single-cell metabolomics to show that Legionella pneumophila infection induces metabolic reprogramming in human macrophages, with bacterial effectors promoting glycolytic shifts.

bioRxiv · Sep 3, 2026 · PreprintMicrobiology

Yersinia pestis lipoprotein SlyB promotes plague pathogenesis via envelope stress tolerance

Pierre Lê-Bury, Emelyne Bougit, Sébastien Bontemps‐Gallo, Guillem Mas Fiol, Cyril Savin, Van Son Nguyen, Julien Madej, Rémi Beau, Caroline Buscail, Nicolas Jean Bouladoux, Friederike Jönsson, COSIPOP study group, Marion Lemarignier, Mara Emilia Carloni, Anne Derbise, , Olivier Dussurget

This study identifies SlyB as a key lipoprotein in Yersinia pestis that enhances envelope stress tolerance, enabling immune evasion in mammalian hosts but not in flea vectors.

bioRxiv · Jun 9, 2026 · PreprintMicrobiology