Author

D. D. Shinde

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

V. C. Thomas· 3 shared A. Stastny· 1 shared B. P. Bertrand· 1 shared C. Rao· 1 shared D. E. Frodyma· 1 shared D. R. Ronning· 1 shared E. Bueno· 1 shared F. Cava· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersMicrobiology

V. C. Thomas

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Staphylococcus aureus counters organic acid anion-mediated inhibition of peptidoglycan cross-linking through robust alanine racemase activity

S. Panda, Y. P. Jayasinghe, D. D. Shinde, E. Bueno, A. Stastny, B. P. Bertrand, S. S. Chaudhari, T. Kielian, F. Cava, D. R. Ronning, V. C. Thomas

This study shows that Staphylococcus aureus counters acetate-mediated inhibition of peptidoglycan cross-linking by maintaining high intracellular D-Ala levels through alanine racemase and limiting D-Ala flux via D-alanine aminotransferase.

bioRxiv · Jan 15, 2024 · PreprintMicrobiology

Host autophagy is exploited by the intracellular parasite Toxoplasma gondii to enhance amino acids levels.

M. D. White, R. K. Angara, L. T. Dias, D. D. Shinde, V. C. Thomas, L. Augusto

This study shows that Toxoplasma gondii exploits host ER-phagy to increase amino acid levels, which supports its persistence and reduces neurological effects in mice.

bioRxiv · Dec 8, 2023 · PreprintMicrobiology

PGC-1β and ERRα promote glutamine metabolism and colorectal cancer survival via transcriptional regulation of PCK2

D. E. Frodyma, T. C. Troia, J. A. Berg, C. Rao, R. A. Svoboda, D. D. Shinde, V. C. Thomas, R. E. Lewis, K. W. Fisher

This study identifies PCK2 as a target of PGC-1β and ERRα that promotes glutamine metabolism and colorectal cancer survival through transcriptional regulation.

bioRxiv · May 20, 2022 · PreprintCancer Biology