Author

D. Dingar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

B. G. Allen· 2 shared J. Trepanier· 2 shared J.-C. Tardif· 2 shared M. G. Sirois· 2 shared M.-A. Gillis· 2 shared P. Sahadevan· 2 shared S. A. Nawaito· 2 shared Y. Shi· 2 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

B. G. Allen

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2 papers

Recent·Most cited

ERK3 Is Involved in Regulating Cardiac Fibroblast Function

P. Sahadevan, D. Dingar, S. A. Nawaito, R. S. Nair, J. Trepanier, F. Sahmi, Y. Shi, M.-A. Gillis, M. G. Sirois, S. Meloche, J.-C. Tardif, B. G. Allen

This study shows ERK3 regulates cardiac fibroblast function by affecting collagen production and motility in response to TGF-β and TAC.

bioRxiv · Dec 5, 2023Cell Biology

MK2-deficient mice are bradycardic and display delayed hypertrophic remodelling in response to a chronic increase in afterload

M. Ruiz, M. Khairallah, D. Dingar, G. Vaniotis, R. J. Khairallah, B. Lauzier, S. Thibault, J. Trepanier, Y. Shi, A. Douillette, B. Hussein, S. A. Nawaito, P. Sahadevan, A. Nguyen, M.-A. Gillis, , B. G. Allen

This study examines the cardiac phenotype of MK2-deficient mice, finding altered autonomic regulation, increased metabolic gene expression, reduced mitochondrial Ca2+-sensitivity, and delayed hypertrophic remodeling in response to pressure overload.

bioRxiv · Jan 23, 2020Physiology