Author

M.-A. Gillis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

B. G. Allen· 3 shared J. Trepanier· 3 shared J.-C. Tardif· 3 shared M. G. Sirois· 3 shared P. Sahadevan· 3 shared S. A. Nawaito· 3 shared Y. Shi· 3 shared D. Dingar· 2 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersPhysiology

B. G. Allen

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ERK3 Is Involved in Regulating Cardiac Fibroblast Function

P. Sahadevan, D. Dingar, S. A. Nawaito, R. S. Nair, J. Trepanier, F. Sahmi, Y. Shi, M.-A. Gillis, M. G. Sirois, S. Meloche, J.-C. Tardif, B. G. Allen

This study shows ERK3 regulates cardiac fibroblast function by affecting collagen production and motility in response to TGF-β and TAC.

bioRxiv · Dec 5, 2023Cell Biology

MK2 deficiency decreases mortality during the inflammatory phase after myocardial infarction in mice

J. Trepanier, S. A. Nawaito, P. Sahadevan, F. Sahmi, N. Duquette, D. Gelinas, M.-A. Gillis, Y. Shi, C. Torok, M.-E. Clavet-Lanthier, M. Gaestel, M. G. Sirois, J.-C. Tardif, B. G. Allen

This study shows MK2 deficiency reduces mortality and alters cytokine levels during the inflammatory phase after myocardial infarction in mice.

bioRxiv · Mar 6, 2023 · PreprintPhysiology

MK2-deficient mice are bradycardic and display delayed hypertrophic remodelling in response to a chronic increase in afterload

M. Ruiz, M. Khairallah, D. Dingar, G. Vaniotis, R. J. Khairallah, B. Lauzier, S. Thibault, J. Trepanier, Y. Shi, A. Douillette, B. Hussein, S. A. Nawaito, P. Sahadevan, A. Nguyen, M.-A. Gillis, , B. G. Allen

This study examines the cardiac phenotype of MK2-deficient mice, finding altered autonomic regulation, increased metabolic gene expression, reduced mitochondrial Ca2+-sensitivity, and delayed hypertrophic remodeling in response to pressure overload.

Journal of the American Heart Association · Feb 16, 2021Peer-reviewedPhysiology