Author

J. Trepanier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

B. G. Allen· 4 shared J.-C. Tardif· 4 shared P. Sahadevan· 4 shared S. A. Nawaito· 4 shared F. Sahmi· 3 shared M. G. Sirois· 3 shared M. Gaestel· 3 shared M.-A. Gillis· 3 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersPhysiology

B. G. Allen

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Author profile →

4 papers

Recent·Most cited

ERK3 Is Involved in Regulating Cardiac Fibroblast Function

P. Sahadevan, D. Dingar, S. A. Nawaito, R. S. Nair, J. Trepanier, F. Sahmi, Y. Shi, M.-A. Gillis, M. G. Sirois, S. Meloche, J.-C. Tardif, B. G. Allen

This study shows ERK3 regulates cardiac fibroblast function by affecting collagen production and motility in response to TGF-β and TAC.

bioRxiv · Dec 5, 2023Cell Biology

MK2 deficiency decreases mortality during the inflammatory phase after myocardial infarction in mice

J. Trepanier, S. A. Nawaito, P. Sahadevan, F. Sahmi, N. Duquette, D. Gelinas, M.-A. Gillis, Y. Shi, C. Torok, M.-E. Clavet-Lanthier, M. Gaestel, M. G. Sirois, J.-C. Tardif, B. G. Allen

This study shows MK2 deficiency reduces mortality and alters cytokine levels during the inflammatory phase after myocardial infarction in mice.

bioRxiv · Mar 6, 2023 · PreprintPhysiology

Expression, subcellular localization, and phosphorylation of MK5 in adult cardiac ventricular fibroblasts

P. Sahadevan, S. A. Nawaito, J. Trepanier, S. Benamar, F. Sahmi, G. Theberge-Julien, L. R. Villeneuve, M. Gaestel, J.-C. Tardif, B. G. Allen

This study investigates MK5 expression, localization, and phosphorylation in cardiac myofibroblasts and myocytes, finding that p38/{beta} mediates T182-MK5 phosphorylation and activation.

bioRxiv · Jul 24, 2020 · PreprintBiochemistry

MK2-deficient mice are bradycardic and display delayed hypertrophic remodelling in response to a chronic increase in afterload

M. Ruiz, M. Khairallah, D. Dingar, G. Vaniotis, R. J. Khairallah, B. Lauzier, S. Thibault, J. Trepanier, Y. Shi, A. Douillette, B. Hussein, S. A. Nawaito, P. Sahadevan, A. Nguyen, M.-A. Gillis, , B. G. Allen

This study examines the cardiac phenotype of MK2-deficient mice, finding altered autonomic regulation, increased metabolic gene expression, reduced mitochondrial Ca2+-sensitivity, and delayed hypertrophic remodeling in response to pressure overload.

bioRxiv · Jan 23, 2020Physiology