Author

D. K. Finlay

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

L. V. Sinclair· 2 shared A. I. I. Lamond· 1 shared A. J. Brenes· 1 shared A. J. M. Howden· 1 shared C. Goggin· 1 shared C. M. Gardiner· 1 shared C. Rollings· 1 shared D. A. Cantrell· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

L. V. Sinclair

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

QUAS-R: Glutamine (Q) Uptake Assay with Single cell Resolution reveals metabolic heterogeneity with immune populations.

L. Pelgrom, G. Davis, S. O Shaughnessy, S. I. Van Kasteren, D. K. Finlay, L. V. Sinclair

The paper introduces a single-cell assay to measure SLC1A5-mediated glutamine uptake, revealing metabolic heterogeneity in immune populations.

bioRxiv · Sep 29, 2022 · PreprintImmunology

The Immunological Proteome Resource

A. J. Brenes, J. L. Hukelmann, L. Spinelli, A. J. M. Howden, J. M. Marchingo, L. V. Sinclair, C. Rollings, O. J. James, I. R. Phair, S. P. Matthews, S. H. Ross, S. C. Arthur, M. Swamy, D. K. Finlay, A. I. I. Lamond, D. A. Cantrell

The paper introduces ImmPRes, a proteomic resource integrating mass-spectrometry data from murine hematopoietic cells to study immune activation and signaling.

bioRxiv · Aug 29, 2022Immunology

TGFβ drives mitochondrial dysfunction in peripheral blood NK cells during metastatic breast cancer

K. Slattery, V. Zaiatz-Bittencourt, E. Woods, K. Brennan, S. Marks, S. Chew, M. Conroy, C. Goggin, J. Kennedy, D. K. Finlay, C. M. Gardiner

This study shows that TGFβ causes mitochondrial dysfunction in NK cells of metastatic breast cancer patients, and blocking TGFβ improves their metabolic function and IFNγ production.

bioRxiv · Jun 2, 2019 · PreprintImmunology