Author

D. Sela-Donenfeld

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Evolutionary Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:chicken / avian· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

R. Kalev-Altman· 2 shared E. Monsonego-Ornan· 1 shared E. Nadjar-Boger· 1 shared H. Wolfenson· 1 shared H. Zhao· 1 shared N. Pokhrel· 1 shared N. Y. Shpigel· 1 shared O. Genin· 1 shared

Activity by Year

2022-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersPhysiology

R. Kalev-Altman

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Author profile →

3 papers

Recent·Most cited

MMP2 loss leads to defective parturition and severe dystocia in mice

R. Kalev-Altman, T. Levy, N. Y. Shpigel, E. Monsonego-Ornan, D. Sela-Donenfeld

This study shows that MMP2 loss causes dystocia and uterine fibrosis in mice, highlighting its role in parturition.

bioRxiv · Nov 22, 2022 · PreprintPhysiology

Initiation of fibronectin fibrillogenesis is an enzyme-dependent process

S. Melamed, S. Zaffryar-Eilot, E. Nadjar-Boger, R. Aviram, H. Zhao, W. Yaseen-Badarne, R. Kalev-Altman, D. Sela-Donenfeld, O. Lewinson, S. Astrof, P. Hasson, H. Wolfenson

This study identifies fibronectin oxidation by lysyl-oxidase enzymes as a force-independent nucleation step initiating fibrillogenesis, which is critical for cell adhesion and mechanosensing.

bioRxiv · Nov 16, 2022Cell Biology

HREM, RNAseq and cell-cycle analyses reveal the role of the G2/M-regulatory protein, Wee1, on the survivability of chicken embryos during diapause.

N. Pokhrel, O. Genin, D. Sela-Donenfeld, Y. Cinnamon

This study uses HREM, RNAseq, and molecular assays to show that Wee1 upregulation during cold diapause arrests the cell cycle, preserving embryonic structure and enabling resumption of development.

bioRxiv · Feb 23, 2022Evolutionary Biology