Author

D. Y. Zhao

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Iqbal· 1 shared F. J. Bauerlein· 1 shared F. Wilfling· 1 shared F.-U. Hartl· 1 shared I. Saha· 1 shared J. F. Greenblatt· 1 shared J. Min· 1 shared K. J. Abraham· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiochemistry

A. Iqbal

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Autophagy preferentially degrades non-fibrillar polyQ aggregates

D. Y. Zhao, F. J. Bauerlein, I. Saha, F.-U. Hartl, W. Baumeister, F. Wilfling

This study shows autophagosomes preferentially degrade non-fibrillar polyQ aggregates by interacting with p62, while amyloid fibrils evade clearance due to their non-deformable structure.

bioRxiv · Aug 8, 2023Cell Biology

A small-molecule antagonist for the Tudor domain of SMN disrupts the interaction between SMN and RNAP II

Y. Liu, A. Iqbal, W. Li, Z. Ni, D. Y. Zhao, K. J. Abraham, W. Tempel, X. Guo, M. Zhou, P. J. Brown, S. Qin, K. Mekhail, J. F. Greenblatt, J. Min

This paper identifies a small-molecule antagonist targeting the Tudor domain of SMN, disrupting its interaction with RNAP II and mimicking SMN depletion effects.

bioRxiv · Mar 2, 2022 · PreprintBiochemistry