Author

Diana Condrea

Centre National de la Recherche Scientifique

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Developmental Biology· 3

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:statistical model· 1

Frequent Co-Authors

Norbert B. GhyselinckiD· 3 shared Betty Féret· 2 shared Chloé MayèreiD· 2 shared Manuel P. MarkiD· 2 shared Muriel Klopfenstein· 2 shared Nadège VernetiD· 2 shared Serge NefiD· 2 shared Violaine Alunni· 2 shared

Activity by Year

2019-2023 · peak 1/yr

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10 top collaborators · 35 mutual co-author links

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3 shared papersDevelopmental Biology

Norbert B. GhyselinckORCID

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3 papers

Recent·Most cited

Loss of NR5A1 in Sertoli cells after sex determination changes their cellular identity and induces their death by anoikis

Sirine Souali‐Crespo, Diana Condrea, Nadège Vernet, Betty Féret, Muriel Klopfenstein, Erwan Grandgirard, Violaine Alunni, Marie Cerciat, Matthieu Jung, Chloé Mayère, Serge Nef, Manuel P. Mark, Frédéric Chalmel, Norbert B. Ghyselinck

This study shows that loss of NR5A1 in Sertoli cells after sex determination leads to their transdifferentiation, anoikis, and testis abnormalities.

bioRxiv · Jan 4, 2023 · PreprintDevelopmental Biology

Origin, specification and differentiation of a rare supporting-like lineage in the developing mouse gonad

Chloé Mayère, Violaine Regard, Aitana Perea-Gómez, Corey Bunce, Yasmine Neirijnck, Cyril Djari, Natividad Bellido-Carreras, Pauline Sararols, Richard Reeves, Simon Greenaway, Michelle Simon, Pam Siggers, Diana Condrea, Françoise Kühne, Ivana Gantar, , Serge Nef

The paper identifies and characterizes a previously uncharacterized supporting-like cell lineage in developing mouse gonads, showing its origin, specification, and sex-specific differentiation.

Science Advances · May 25, 2022Peer-reviewedDevelopmental Biology

Meiosis Initiates In The Fetal Ovary Of Mice Lacking All Retinoic Acid Receptor Isotypes

Nadège Vernet, Manuel P. Mark, Diana Condrea, Betty Féret, Muriel Klopfenstein, Violaine Alunni, Marius Teletin, Norbert B. Ghyselinck

This study shows that retinoic acid receptors are not required for meiosis initiation in mouse fetal ovaries, challenging the current model and suggesting a role for CYP26B1 in meiosis prevention.

bioRxiv · Jul 26, 2019 · PreprintDevelopmental Biology