Author

E. Bjanes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. A. DeLaney· 1 shared A. B. Janssen· 1 shared A. M. Bravo· 1 shared A. Stream· 1 shared A. Varble· 1 shared B. C. Schaefer· 1 shared B. Demarco· 1 shared B. L. Lee· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. A. DeLaney

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

An efficient in vivo-inducible CRISPR interference system for group A Streptococcus genetic analysis and pathogenesis studies

E. Bjanes, A. Stream, A. B. Janssen, P. S. Gibson, A. M. Bravo, S. Dahesh, J. L. Baker, A. Varble, V. Nizet, J.-W. Veening

This paper develops a doxycycline-inducible CRISPRi system for GAS to enable efficient gene repression and presents a genome browser for GAS genetic features.

bioRxiv · Feb 22, 2024 · PreprintMicrobiology

Genetic targeting of Card19 is linked to disrupted Ninj1 expression, impaired cell lysis, and increased susceptibility to Yersinia infection

E. Bjanes, R. G. Sillas, R. Matsuda, B. Demarco, T. Fettrelet, A. A. DeLaney, O. S. Kornfeld, B. L. Lee, E. M. Rodriguez Lopez, D. Grubaugh, M. A. Wynosky-Dolfi, N. H. Philip, E. Krespan, D. Tovar, L. Joannas, , I. E. Brodsky

This study shows that genetic targeting of Card19 disrupts Ninj1 expression, impairs macrophage cell lysis, and increases susceptibility to Yersinia infection.

bioRxiv · Mar 19, 2021 · PreprintImmunology