Author

E. Krespan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

D. P. Beiting· 2 shared I. E. Brodsky· 2 shared R. Matsuda· 2 shared A. A. DeLaney· 1 shared B. C. Schaefer· 1 shared B. Demarco· 1 shared B. L. Lee· 1 shared C.-A. Assenmacher· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersMicrobiology

D. P. Beiting

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Inflammatory monocytes promote pyogranuloma formation to counteract Yersinia blockade of host defense

D. Sorobetea, R. Matsuda, S. T. Peterson, J. P. Grayczyk, I. Rao, E. Krespan, M. Lanza, C.-A. Assenmacher, D. P. Beiting, E. Radaelli, I. E. Brodsky

This study shows that Yersinia pseudotuberculosis induces pyogranulomas in the murine intestine by blocking phagocytosis, and that monocytes are essential for granuloma formation and immune control.

bioRxiv · Feb 6, 2022 · PreprintMicrobiology

Genetic targeting of Card19 is linked to disrupted Ninj1 expression, impaired cell lysis, and increased susceptibility to Yersinia infection

E. Bjanes, R. G. Sillas, R. Matsuda, B. Demarco, T. Fettrelet, A. A. DeLaney, O. S. Kornfeld, B. L. Lee, E. M. Rodriguez Lopez, D. Grubaugh, M. A. Wynosky-Dolfi, N. H. Philip, E. Krespan, D. Tovar, L. Joannas, , I. E. Brodsky

This study shows that genetic targeting of Card19 disrupts Ninj1 expression, impairs macrophage cell lysis, and increases susceptibility to Yersinia infection.

bioRxiv · Mar 19, 2021 · PreprintImmunology