Author

E. Christodoulou-Vafeiadou

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

G. KOLLIAS· 3 shared L. NTARI· 3 shared N. KARAGIANNI· 3 shared C. GEKA· 2 shared M. C. DENIS· 2 shared A. N. Matralis· 1 shared A.-K. Pedersen· 1 shared C. Pantos· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersImmunology

G. KOLLIAS

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivNov 8, 2023

A Novel human IL-23A Overexpressing Mouse Model of Systemic Lupus Erythematosus

E. CHRISTODOULOU-VAFEIADOU, C. GEKA, L. ILIOPOULOU…·G. KOLLIAS

This paper creates a human IL-23A overexpressing mouse model of SLE and shows it develops autoimmune features similar to human SLE.

DOI 10.1002/art.42830

mouseexperimental assay

𝕏 PostBluesky

Pharmacology & ToxicologybioRxivPreprintAug 2, 2022

Repurposing of Amisulpride, a known antipsychotic drug, to target synovial fibroblasts activation in arthritis.

D. Papadopoulou, F. Roumelioti, C. Tzaferis…·G. Kollias

This study identifies Amisulpride as a potential therapeutic for arthritis by reducing synovial fibroblast activation, independent of its known targets.

DOI 10.1101/2022.08.02.500956

mouseproteomics

𝕏 PostBluesky

PathologybioRxivMay 14, 2020

Ectopic bone formation and systemic bone loss in a transmembrane TNF-driven model of human spondyloarthritis

E. Christodoulou-Vafeiadou, C. Geka, L. Ntari…·G. Kollias

This paper characterizes a transmembrane TNF-driven mouse model of spondyloarthritis, showing ectopic bone formation, systemic bone loss, and comorbid heart valve pathology, with early anti-TNF treatment reducing inflammation.

DOI 10.1186/s13075-020-02327-4

𝕏 PostBluesky