Author

L. Ntari

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

E. CHRISTODOULOU-VAFEIADOU· 3 shared G. KOLLIAS· 3 shared N. KARAGIANNI· 3 shared C. GEKA· 2 shared M. C. DENIS· 2 shared A. N. Matralis· 1 shared A.-K. Pedersen· 1 shared C. Pantos· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersImmunology

E. CHRISTODOULOU-VAFEIADOU

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Author profile →

3 papers

Recent·Most cited

A Novel human IL-23A Overexpressing Mouse Model of Systemic Lupus Erythematosus

E. CHRISTODOULOU-VAFEIADOU, C. GEKA, L. ILIOPOULOU, L. NTARI, M. C. DENIS, N. KARAGIANNI, G. KOLLIAS

This paper creates a human IL-23A overexpressing mouse model of SLE and shows it develops autoimmune features similar to human SLE.

bioRxiv · Nov 8, 2023Immunology

Repurposing of Amisulpride, a known antipsychotic drug, to target synovial fibroblasts activation in arthritis.

D. Papadopoulou, F. Roumelioti, C. Tzaferis, P. Chouvardas, A.-K. Pedersen, F. Charalampous, E. Christodoulou-Vafeiadou, L. Ntari, N. Karagianni, M. Denis, J. V. Olsen, A. N. Matralis, G. Kollias

This study identifies Amisulpride as a potential therapeutic for arthritis by reducing synovial fibroblast activation, independent of its known targets.

bioRxiv · Aug 2, 2022 · PreprintPharmacology & Toxicology

Ectopic bone formation and systemic bone loss in a transmembrane TNF-driven model of human spondyloarthritis

E. Christodoulou-Vafeiadou, C. Geka, L. Ntari, K. Kranidioti, E. Argyropoulou, F. Meier, M. Armaka, I. Mourouzis, C. Pantos, M. Rouchota, G. Loudos, M. C. Denis, N. Karagianni, G. Kollias

This paper characterizes a transmembrane TNF-driven mouse model of spondyloarthritis, showing ectopic bone formation, systemic bone loss, and comorbid heart valve pathology, with early anti-TNF treatment reducing inflammation.

bioRxiv · May 14, 2020Pathology