Author

E. Hadadi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Debraekeleer· 2 shared D. Kancheva· 2 shared D. Laoui· 2 shared J. Brughmans· 2 shared M. Kiss· 2 shared Y. Elkrim· 2 shared A. Bennaceur-Griscelli· 1 shared A. D. Garg· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersImmunology

A. Debraekeleer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Epigenomic programming of peripheral monocytes determines their transcriptional response to the tumor microenvironment

M. Kiss, L. Halasz, E. Hadadi, W. K. Berger, P. Tzerpos, S. Poliska, D. Kancheva, A. Debraekeleer, J. Brughmans, Y. Elkrim, L. Martens, Y. Saeys, B. Daniel, Z. Czimmerer, D. Laoui, , J. A. Van Ginderachter

This study shows that peripheral monocytes' epigenomic landscape is rewired by a distal tumor, shaping their transcriptional response upon tumor infiltration.

bioRxiv · Jun 19, 2024Immunology

Distinct cDC subsets co-operate in CD40 agonist response while suppressive microenvironments and lack of antigens subvert efficacy

A. Murgaski, M. Kiss, H. Van Damme, D. Kancheva, I. Vanmeerbeek, J. Keirsse, E. Hadadi, J. Brughmans, S. M. Arnouk, A. E. I. Hamouda, A. Debraekeleer, V. Bosteels, Y. Elkrim, L. Boon, S. Hoves, , D. Laoui

This study identifies cDC subsets that cooperate in CD40 agonist response and shows that suppressive microenvironments and antigen scarcity limit its efficacy in cancer.

bioRxiv · Dec 25, 2021 · PreprintCancer Biology

Chronic circadian disruption modulates breast cancer cell stemness and their immune microenvironment to drive metastasis in mice

E. Hadadi, W. Taylor, X. Li, Y. Aslan, M. Vilotte, J. Riviere, G. Duvallet, C. Auriau, S. Dulong, I. Raymond Letron, S. Provot, A. Bennaceur-Griscelli, H. Acloque

This study shows chronic circadian disruption in mice increases breast cancer metastasis by enhancing stemness and creating an immunosuppressive microenvironment, which can be reversed by a CXCR2 inhibitor.

bioRxiv · Sep 26, 2019Cancer Biology