Author

E. N. Cytrynbaum

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Biophysics· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 4 Research Focus:cell signaling & trafficking· 3 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Bautista· 1 shared B. Gablaski· 1 shared C. B. Macdonald· 1 shared C. Cras-Meneur· 1 shared C. E. Ellis· 1 shared C. Nislow· 1 shared D. A. Dionne· 1 shared D. Hutchinson· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperPhysiology

A. Bautista

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Author profile →

4 papers

Recent·Most cited

A stochastic model of cortical microtubule anchoring and mechanics provides regulatory control of microtubule shape

T. Y. Y. Tian, C. B. Macdonald, E. N. Cytrynbaum

The paper develops a stochastic model of cortical microtubule anchoring and mechanics to show how tuning growth speed to anchoring rate can prevent buckling and deflection in plant cells.

bioRxiv · Sep 8, 2022Biophysics

A Multiscale computational model of YAP signaling in epithelial fingering behaviour

N. Mukhtar, L. Edelstein-Keshet, E. N. Cytrynbaum

The paper presents a multiscale computational model of YAP signaling in epithelial cells, showing how it drives EMT and fingering behavior under topographic cues.

bioRxiv · Dec 13, 2021Cell Biology

The mechanism of MinD stability modulation by MinE in Min protein dynamics

W. C. Carlquist, E. N. Cytrynbaum

The paper fits mathematical models to time-course Min protein concentration data to determine the mechanism of MinD stability modulation by MinE.

bioRxiv · Jul 30, 2021Biochemistry

Beta-cell specific Insr deletion promotes insulin hypersecretion and improves glucose tolerance prior to global insulin resistance

S. Skovso, E. Panzhinskiy, J. Kolic, H. H. Cen, D. A. Dionne, X.-Q. Dai, R. B. Sharma, L. Elghazi, C. E. Ellis, K. Faulkner, S. A. M. Marcil, P. Overby, N. Noursadeghi, D. Hutchinson, X. Hu, , J. D. Johnson

This study shows that deleting the insulin receptor in beta-cells improves glucose tolerance in some mice but not others, depending on age and diet.

bioRxiv · Oct 15, 2020Physiology