Author

E. P. Sulman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. P. Chumakova· 1 shared A. Poteete· 1 shared C. Danussi· 1 shared F. M. Johnson· 1 shared F. Zhang· 1 shared G. J. Riggins· 1 shared J. D. Lathia· 1 shared J. Heymach· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. P. Chumakova

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Author profile →

3 papers

Recent·Most cited

Inhibition of nonsense-mediated decay rescues functional p53β/γ isoforms in MDM2-amplified cancers

J. P. Gudikote, T. Cascone, A. Poteete, P. Sitthideatphaiboon, Q. Wu, N. Morikawa, F. Zhang, S. Peng, P. Tong, L. Li, L. Shen, M. Nilsson, P. Jones, E. P. Sulman, J. Wang, , J. Heymach

The paper shows that inhibiting nonsense-mediated decay increases expression of p53β/γ isoforms, which are resistant to MDM2 degradation, thereby restoring p53 function in cancers with MDM2 amplification, HPV infection, or specific p53 mutations.

bioRxiv · Apr 6, 2020 · PreprintCancer Biology

High-throughput Automated Single Cell Imaging Analysis Reveals Dynamics Of Glioblastoma Stem Cell Population During State Transition

A. P. Chumakova, M. Hitomi, E. P. Sulman, J. D. Lathia

The paper develops HASCIA, a high-throughput automated single-cell imaging analysis tool, to quantify protein expression and study dynamics of glioblastoma stem cell state transitions.

bioRxiv · Jul 31, 2018Cancer Biology

G-quadruplex DNA drives genomic instability and represents a targetable molecular abnormality in ATRX-deficient malignant glioma

Y. Wang, J. Yang, W. H. Wu, R. Shah, C. Danussi, G. J. Riggins, K. Kannan, E. P. Sulman, T. A. Chan, J. T. Huse

This study shows ATRX deficiency in glioma causes genomic instability via G-quadruplex DNA, and G4 stabilization can be therapeutically targeted.

bioRxiv · Jun 14, 2018Cancer Biology